human kidney proximal tubule epithelial cells hk 2 (Procell Inc)
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Procell Inc
human kidney proximal tubule epithelial cells hk 2
Human Kidney Proximal Tubule Epithelial Cells Hk 2, supplied by Procell Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+kidney+proximal+tubule+epithelial+cells+(hk-2)/cells+hek293t/pmc12957774-54-0-8
Average 86 stars, based on 1 article reviews
Human Kidney Proximal Tubule Epithelial Cells Hk 2, supplied by Procell Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+kidney+proximal+tubule+epithelial+cells+(hk-2)/cells+hek293t/pmc12957774-54-0-8
Average 86 stars, based on 1 article reviews
human kidney proximal tubule epithelial cells hk 2 - by Bioz Stars,
2026-10
86/100 stars
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Cell Culture:Article Title: Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury Article Snippet: Article Title: Granulin in renal tubular epithelia is associated with interstitial inflammation and activates the TLR9-IFN-α pathway in lupus nephritis. Article Snippet: Objective: This study aimed to investigate the possible role of granulin (GRN) in activating the TLR9-IFN-α pathway in renal tubular epithelial cells (RTECs) and explore clues that RTECs regulate the micro-environment of inflammatory response in lupus nephritis (LN).. Methods: Renal sections from 57 LN patients and 30 non-LN patients were sampled for histological study, and GRN overexpression RTECs were applied for cytological study.. Results: In the histological study, GRN is highly expressed in LN RTECs with tubulointerstitial inflammation (TII) and well co-localized with TLR9. Article Title: Silencing of adipocyte enhancer-binding protein 1 (AEBP1) alleviates renal fibrosis in vivo and in vitro via inhibition of the β-catenin signaling pathway. Article Snippet: Renal fibrosis is the common final pathway in many renal diseases regardless of the underlying etiology.. Adipocyte enhancerbinding protein 1 (AEBP1) was reported to play a vital role in the development of organ fibrosis, but its role in renal fibrosis has not been reported.. Thus, the aim of this study was to investigate the possible function of AEBP1 in renal fibrosis and the mechanism associated with the β-catenin signaling pathway. Article Title: Cytosolic mtDNA-cGAS-STING axis contributes to sepsis-induced acute kidney injury via activating the NLRP3 inflammasome. Article Snippet: Background NLRP3 inflammasome activation is significantly associated with sepsis-induced acute kidney injury (S-AKI).. Cytosolic DNA derived from damaged mitochondria has been reported to activate NLRP3 inflammasome via upregulating the cyclic GMP–AMP synthase (cGAS)—the stimulator of interferon genes (STING) axis in nucleus pulposus cell and cardiomyocytes.. However, the regulatory effect of mitochondria DNA (mtDNA)–cGAS–STING axis on the NLRP3 inflammasome in S-AKI remains unclear. Modification:Article Title: Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury Article Snippet: Article Title: Granulin in renal tubular epithelia is associated with interstitial inflammation and activates the TLR9-IFN-α pathway in lupus nephritis. Article Snippet: Objective: This study aimed to investigate the possible role of granulin (GRN) in activating the TLR9-IFN-α pathway in renal tubular epithelial cells (RTECs) and explore clues that RTECs regulate the micro-environment of inflammatory response in lupus nephritis (LN).. Methods: Renal sections from 57 LN patients and 30 non-LN patients were sampled for histological study, and GRN overexpression RTECs were applied for cytological study.. Results: In the histological study, GRN is highly expressed in LN RTECs with tubulointerstitial inflammation (TII) and well co-localized with TLR9. Article Title: Silencing of adipocyte enhancer-binding protein 1 (AEBP1) alleviates renal fibrosis in vivo and in vitro via inhibition of the β-catenin signaling pathway. Article Snippet: Renal fibrosis is the common final pathway in many renal diseases regardless of the underlying etiology.. Adipocyte enhancerbinding protein 1 (AEBP1) was reported to play a vital role in the development of organ fibrosis, but its role in renal fibrosis has not been reported.. Thus, the aim of this study was to investigate the possible function of AEBP1 in renal fibrosis and the mechanism associated with the β-catenin signaling pathway. Article Title: Cytosolic mtDNA-cGAS-STING axis contributes to sepsis-induced acute kidney injury via activating the NLRP3 inflammasome. Article Snippet: Background NLRP3 inflammasome activation is significantly associated with sepsis-induced acute kidney injury (S-AKI).. Cytosolic DNA derived from damaged mitochondria has been reported to activate NLRP3 inflammasome via upregulating the cyclic GMP–AMP synthase (cGAS)—the stimulator of interferon genes (STING) axis in nucleus pulposus cell and cardiomyocytes.. However, the regulatory effect of mitochondria DNA (mtDNA)–cGAS–STING axis on the NLRP3 inflammasome in S-AKI remains unclear. Control:Article Title: Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury Article Snippet: Article Title: Granulin in renal tubular epithelia is associated with interstitial inflammation and activates the TLR9-IFN-α pathway in lupus nephritis. Article Snippet: Objective: This study aimed to investigate the possible role of granulin (GRN) in activating the TLR9-IFN-α pathway in renal tubular epithelial cells (RTECs) and explore clues that RTECs regulate the micro-environment of inflammatory response in lupus nephritis (LN).. Methods: Renal sections from 57 LN patients and 30 non-LN patients were sampled for histological study, and GRN overexpression RTECs were applied for cytological study.. Results: In the histological study, GRN is highly expressed in LN RTECs with tubulointerstitial inflammation (TII) and well co-localized with TLR9. Article Title: Silencing of adipocyte enhancer-binding protein 1 (AEBP1) alleviates renal fibrosis in vivo and in vitro via inhibition of the β-catenin signaling pathway. Article Snippet: Renal fibrosis is the common final pathway in many renal diseases regardless of the underlying etiology.. Adipocyte enhancerbinding protein 1 (AEBP1) was reported to play a vital role in the development of organ fibrosis, but its role in renal fibrosis has not been reported.. Thus, the aim of this study was to investigate the possible function of AEBP1 in renal fibrosis and the mechanism associated with the β-catenin signaling pathway. Article Title: Cytosolic mtDNA-cGAS-STING axis contributes to sepsis-induced acute kidney injury via activating the NLRP3 inflammasome. Article Snippet: Background NLRP3 inflammasome activation is significantly associated with sepsis-induced acute kidney injury (S-AKI).. Cytosolic DNA derived from damaged mitochondria has been reported to activate NLRP3 inflammasome via upregulating the cyclic GMP–AMP synthase (cGAS)—the stimulator of interferon genes (STING) axis in nucleus pulposus cell and cardiomyocytes.. However, the regulatory effect of mitochondria DNA (mtDNA)–cGAS–STING axis on the NLRP3 inflammasome in S-AKI remains unclear. Incubation:Article Title: Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury Article Snippet: Article Title: Granulin in renal tubular epithelia is associated with interstitial inflammation and activates the TLR9-IFN-α pathway in lupus nephritis. Article Snippet: Objective: This study aimed to investigate the possible role of granulin (GRN) in activating the TLR9-IFN-α pathway in renal tubular epithelial cells (RTECs) and explore clues that RTECs regulate the micro-environment of inflammatory response in lupus nephritis (LN).. Methods: Renal sections from 57 LN patients and 30 non-LN patients were sampled for histological study, and GRN overexpression RTECs were applied for cytological study.. Results: In the histological study, GRN is highly expressed in LN RTECs with tubulointerstitial inflammation (TII) and well co-localized with TLR9. Article Title: Silencing of adipocyte enhancer-binding protein 1 (AEBP1) alleviates renal fibrosis in vivo and in vitro via inhibition of the β-catenin signaling pathway. Article Snippet: Renal fibrosis is the common final pathway in many renal diseases regardless of the underlying etiology.. Adipocyte enhancerbinding protein 1 (AEBP1) was reported to play a vital role in the development of organ fibrosis, but its role in renal fibrosis has not been reported.. Thus, the aim of this study was to investigate the possible function of AEBP1 in renal fibrosis and the mechanism associated with the β-catenin signaling pathway. Article Title: Cytosolic mtDNA-cGAS-STING axis contributes to sepsis-induced acute kidney injury via activating the NLRP3 inflammasome. Article Snippet: Background NLRP3 inflammasome activation is significantly associated with sepsis-induced acute kidney injury (S-AKI).. Cytosolic DNA derived from damaged mitochondria has been reported to activate NLRP3 inflammasome via upregulating the cyclic GMP–AMP synthase (cGAS)—the stimulator of interferon genes (STING) axis in nucleus pulposus cell and cardiomyocytes.. However, the regulatory effect of mitochondria DNA (mtDNA)–cGAS–STING axis on the NLRP3 inflammasome in S-AKI remains unclear. Marker:Article Title: Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury Article Snippet: Article Title: Granulin in renal tubular epithelia is associated with interstitial inflammation and activates the TLR9-IFN-α pathway in lupus nephritis. Article Snippet: Objective: This study aimed to investigate the possible role of granulin (GRN) in activating the TLR9-IFN-α pathway in renal tubular epithelial cells (RTECs) and explore clues that RTECs regulate the micro-environment of inflammatory response in lupus nephritis (LN).. Methods: Renal sections from 57 LN patients and 30 non-LN patients were sampled for histological study, and GRN overexpression RTECs were applied for cytological study.. Results: In the histological study, GRN is highly expressed in LN RTECs with tubulointerstitial inflammation (TII) and well co-localized with TLR9. Article Title: Silencing of adipocyte enhancer-binding protein 1 (AEBP1) alleviates renal fibrosis in vivo and in vitro via inhibition of the β-catenin signaling pathway. Article Snippet: Renal fibrosis is the common final pathway in many renal diseases regardless of the underlying etiology.. Adipocyte enhancerbinding protein 1 (AEBP1) was reported to play a vital role in the development of organ fibrosis, but its role in renal fibrosis has not been reported.. Thus, the aim of this study was to investigate the possible function of AEBP1 in renal fibrosis and the mechanism associated with the β-catenin signaling pathway. Article Title: Cytosolic mtDNA-cGAS-STING axis contributes to sepsis-induced acute kidney injury via activating the NLRP3 inflammasome. Article Snippet: Background NLRP3 inflammasome activation is significantly associated with sepsis-induced acute kidney injury (S-AKI).. Cytosolic DNA derived from damaged mitochondria has been reported to activate NLRP3 inflammasome via upregulating the cyclic GMP–AMP synthase (cGAS)—the stimulator of interferon genes (STING) axis in nucleus pulposus cell and cardiomyocytes.. However, the regulatory effect of mitochondria DNA (mtDNA)–cGAS–STING axis on the NLRP3 inflammasome in S-AKI remains unclear. Isolation:Article Title: Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury Article Snippet: Article Title: Granulin in renal tubular epithelia is associated with interstitial inflammation and activates the TLR9-IFN-α pathway in lupus nephritis. Article Snippet: Objective: This study aimed to investigate the possible role of granulin (GRN) in activating the TLR9-IFN-α pathway in renal tubular epithelial cells (RTECs) and explore clues that RTECs regulate the micro-environment of inflammatory response in lupus nephritis (LN).. Methods: Renal sections from 57 LN patients and 30 non-LN patients were sampled for histological study, and GRN overexpression RTECs were applied for cytological study.. Results: In the histological study, GRN is highly expressed in LN RTECs with tubulointerstitial inflammation (TII) and well co-localized with TLR9. Article Title: Silencing of adipocyte enhancer-binding protein 1 (AEBP1) alleviates renal fibrosis in vivo and in vitro via inhibition of the β-catenin signaling pathway. Article Snippet: Renal fibrosis is the common final pathway in many renal diseases regardless of the underlying etiology.. Adipocyte enhancerbinding protein 1 (AEBP1) was reported to play a vital role in the development of organ fibrosis, but its role in renal fibrosis has not been reported.. Thus, the aim of this study was to investigate the possible function of AEBP1 in renal fibrosis and the mechanism associated with the β-catenin signaling pathway. Article Title: Cytosolic mtDNA-cGAS-STING axis contributes to sepsis-induced acute kidney injury via activating the NLRP3 inflammasome. Article Snippet: Background NLRP3 inflammasome activation is significantly associated with sepsis-induced acute kidney injury (S-AKI).. Cytosolic DNA derived from damaged mitochondria has been reported to activate NLRP3 inflammasome via upregulating the cyclic GMP–AMP synthase (cGAS)—the stimulator of interferon genes (STING) axis in nucleus pulposus cell and cardiomyocytes.. However, the regulatory effect of mitochondria DNA (mtDNA)–cGAS–STING axis on the NLRP3 inflammasome in S-AKI remains unclear. |
